Roles of astrocytic Connexin 43 gap junctional deficit in alpha-synucleinopathy and inflammation relevant to Parkinson’s
Experimental Neurology (2026), 115897. DOI: 10.1016/j.expneurol.2026.115897.
Pro-repair properties of a human embryonic stem cell-derived astrocyte cell therapy in demyelinating disorders
Stem Cell Reports (2026), (June, 102960). DOI: 10.1016/j.stemcr.2026.102960
Online aptamer-based solid-phase extraction coupled with high-performance liquid chromatography–mass spectrometry for the determination of Alzheimer's disease biomarkers
Analytica Chimica Acta, (2026), (Vol. 1417). DOI: 10.1016/j.aca.2026.345809.
Peptide-mediated inhibition of aberrant chaperone-mediated autophagy in pericytes prevents glioblastoma progression through MAPT/tau secretion
Autophagy (2026), 1–26. DOI: 10.1080/15548627.2026.2672700
PARP1 deficiency mitigates amyloid pathology, neurodegeneration, and cognitive decline in a familial Alzheimer’s disease model
PNAS (2026), (Vol. 123, 20). DOI: 10.1073/pnas.2525028123
NMR characterization of the structure and interaction of an RNA aptamer targeting α-synuclein
Biochemical and Biophysical Research Communications (2026), (153874). DOI: 10.1016/j.bbrc.2026.153874.
Microfluidic compartmentalization reveals that ferrostatin-1 restores directional mitochondrial transport in Aβ-challenged neurons
Royal Society of Chemistry (2026). DOI: 10.1039/D5LC00834D
von Willebrand Factor fibers formed at pathological high shear provide a scaffold for α-synuclein binding and aggregation
Nature, Sci Rep (2026). DOI: 10.1038/s41598-026-50190-1
Quantitative biochemical pro ling of GCase activity and α-synuclein proteoforms in postmortem human brains from GBA-related and idiopathic Parkinson’s disease
Research Square (2026). DOI: unavailable.
Deletion of SPI1 in microglia exacerbates amyloid pathology by impairing microglial response in Alzheimer’s disease models
Cell (2026). DOI: 10.1016/j.neuron.2026.03.011