Peptide-mediated inhibition of aberrant chaperone-mediated autophagy in pericytes prevents glioblastoma progression through MAPT/tau secretion
Autophagy (2026), 1–26. DOI: 10.1080/15548627.2026.2672700
PARP1 deficiency mitigates amyloid pathology, neurodegeneration, and cognitive decline in a familial Alzheimer’s disease model
PNAS (2026), (Vol. 123, 20). DOI: 10.1073/pnas.2525028123
NMR characterization of the structure and interaction of an RNA aptamer targeting α-synuclein
Biochemical and Biophysical Research Communications (2026), (153874). DOI: 10.1016/j.bbrc.2026.153874.
Microfluidic compartmentalization reveals that ferrostatin-1 restores directional mitochondrial transport in Aβ-challenged neurons
Royal Society of Chemistry (2026). DOI: 10.1039/D5LC00834D
von Willebrand Factor fibers formed at pathological high shear provide a scaffold for α-synuclein binding and aggregation
Nature, Sci Rep (2026). DOI: 10.1038/s41598-026-50190-1
Quantitative biochemical pro ling of GCase activity and α-synuclein proteoforms in postmortem human brains from GBA-related and idiopathic Parkinson’s disease
Research Square (2026). DOI: unavailable.
Deletion of SPI1 in microglia exacerbates amyloid pathology by impairing microglial response in Alzheimer’s disease models
Cell (2026). DOI: 10.1016/j.neuron.2026.03.011
Tau catalyzes amyloid-β aggregation and toxicity in a polymorph-dependent manner
PNAS (2026), (Vol. 123, 13). DOI: 10.1073/pnas.2532775123
Protocol for quantifying the oxidative cleavage of amyloid-β using oxygen-18 isotope labeling and LC-MS/MS
STAR Protocols (2026), (Vol 7, 1). DOI: 10.1016/j.xpro.2026.104413.
High-pH NMR to Identify Macromolecular Hydrogen-Bonds and Foldons
bioRxiv (2026). DOI: 10.64898/2026.02.28.708709