Rattle-Structured Upconversion Nanoparticles for Near-IR-Induced Suppression of Alzheimer's β-Amyloid Aggregation
Small; DOI:10.1002/smll.201603139.
Changes in lipid membranes may trigger amyloid toxicity in Alzheimer's disease.
PLOS One; doi: 10.1371/journal.pone.0182194.
Amyloidogenicity and toxicity of the reverse and scrambled variants of amyloid‐β 1‐42.
FEBS Letters; https://doi.org/10.1002/1873-3468.12590.
Covalent modifications of the amyloid beta peptide by hydroxynonenal: Effects on metal ion binding by monomers and insights into the fibril topology.
Journal of Inorganic Biochemistry; DOI: 10.1016/j.jinorgbio.2017.06.007.
Carbon Nanodot‐Sensitized Modulation of Alzheimer's β‐Amyloid Self‐Assembly, Disassembly, and Toxicity.
Small; doi: 10.1002/smll.201700983.
PROGRESS ON THE DEVELOPMENT OF CERTIFIED REFERENCE MATERIALS FOR Aβ1-42.
Journal of the Alzheimer’s Association; https://doi.org/10.1016/j.jalz.2017.07.629.
Coconut oil protects cortical neurons from amyloid beta toxicity by enhancing signaling of cell survival pathways.
Science Direct Neurochemistry International; http://dx.doi.org/10.1016/j.neuint.2017.01.008.
Assessment of Novel Curcumin Derivatives as Potent Inhibitors of Inflammation and Amyloid-β Aggregation in Alzheimer’s Disease.
Journal of Alzheimer’s Disease; doi: 10.3233/JAD-170071.
Microglial Activation Results in Inhibition of TGF-β-Regulated Gene Expression.
J Mol Neurosci.; doi: 10.1007/s12031-017-0980-3.
Inhibition of PDE2 reverses beta amyloid induced memory impairment through regulation of PKA/PKG-dependent neuro-inflammatory and apoptotic pathways.
Scientific Reports; https://doi.org/10.1038/s41598-017-08070-2.