Apolipoprotein E Isoforms Differentially Regulate Alzheimer’s Disease and Amyloid-β-Induced Inflammatory Response in vivo and in vitro.
Journal of Alzheimer’s Disease; DOI: 10.3233/JAD-160133.
Potential Artifacts in Sample Preparation Methods Used for Imaging Amyloid Oligomers and Protofibrils due to Surface-Mediated Fibril Formation.
Journal of Physical Chemistry; https://doi.org/10.1021/acs.jpcb.6b12560.
Unmodified and pyroglutamylated amyloid β peptides form hypertoxic hetero-oligomers of unique secondary structure
The Febs Journal; 10.1111/febs.14058.
Dissecting the behaviour of β‐amyloid peptide variants during oligomerization and fibrillation.
Journal of Peptide Science; doi: 10.1002/psc.3028.
Kalirin Reduction Rescues Psychosis-Associated Behavioral Deficits in APPswe/PSEN1dE9 Transgenic Mice.
Neurobiology of Aging; http://dx.doi.org/10.1016/j.neurobiolaging.2017.02.006.
CD44 Splice Variants as Potential Players in Alzheimer’s Disease Pathology.
Journal of Alzheimer’s Disease; doi: 10.3233/JAD-161245.
In situ fibrillizing amyloid-beta 1-42 induces neurite degeneration and apoptosis of differentiated SH-SY5Y cells.
PLOS ONE; doi: 10.1371/journal.pone.0186636.
Analysis of the Impact of CD200 on Phagocytosis.
Mol Neurobiol.; doi: 10.1007/s12035-016-0223-6.
Role of microglial amylin receptors in mediating beta amyloid (Aβ)-induced inflammation.
Journal of Neuroinflammation; https://doi.org/10.1186/s12974-017-0972-9.
Application of quinazoline and pyrido[3,2-d]pyrimidine templates to design multi-targeting agents in Alzheimer's disease
RSC Advances; DOI: 10.1039/C7RA02889J.